jet

joined 2 years ago
MODERATOR OF
[–] jet@hackertalks.com 2 points 2 weeks ago

https://en.wikipedia.org/wiki/How_to_Lie_with_Statistics

I remember reading this book when i was young and impressionable, valuable life lesson

[–] jet@hackertalks.com 2 points 2 weeks ago (1 children)

if you want an example of his "persona" I can think of no better video then this: https://youtu.be/BVbs6gux5vE

[–] jet@hackertalks.com 2 points 2 weeks ago (1 children)

kidney stones are calcium oxalates (typically). I'm glad you have solved kidney stones! Just drink more water.

[–] jet@hackertalks.com 2 points 2 weeks ago

Correct! glucose and vitamin c both compete with the GLUT-4 transporter.... if you dont have excessive glucose, then you don't have excessive competition. This is why the zero-carb diet avoids scurvy!

[–] jet@hackertalks.com 2 points 2 weeks ago

Yeah... that is how big empires fall I suppose. complacency

[–] jet@hackertalks.com 2 points 2 weeks ago* (last edited 2 weeks ago) (3 children)

she isn't selling supplements, just the recommendation to avoid oxalates in your food.

If you want paperwork:

paperworkhttps://pubmed.ncbi.nlm.nih.gov/?term=oxalate+symptoms

If you don't want to buy her book, or if your too lazy to pirate it, and you can't be bothered to watch the interview.... I'll give you the TLDR: Oxalates kinda suck, but most people don't care until they get a kidney stone. If you are concerned then don't eat food with oxalates in it.

[–] jet@hackertalks.com 2 points 2 weeks ago* (last edited 2 weeks ago) (2 children)

video summerizer

Summary

In this detailed and informative interview on the Low Carb Ancestral Living channel, host Pym Johnson revisits the topic of oxalates with expert Sally K. Norton, a well-known advocate and researcher on oxalate toxicity and healing. The conversation delves deep into the chemistry, physiology, and health implications of oxalates—naturally occurring compounds in many plants—and their impact on human health, especially in relation to chronic diseases, kidney stones, and systemic inflammation. Sally explains the dual nature of oxalates as acids and salts, their formation of nano- and micro-crystals in the body, and how these crystals can accumulate in various tissues causing oxidative stress, inflammation, and damage to organs such as kidneys, bones, joints, and glands.

The discussion also covers which foods are high in oxalates, including popular leafy greens, nuts, seeds, grains, and certain fruits like kiwi and raspberries, while emphasizing the importance of avoiding these for those sensitive or poisoned by oxalates. Sally explains why some plant foods historically considered healthy can be problematic due to their oxalate content, and shares practical advice on managing oxalate intake, including food preparation techniques and dietary choices.

Sally further discusses symptoms linked with oxalate poisoning, which range from joint pain, arthritis, skin issues, fungal infections, fatigue, migraines, neurological symptoms, to pelvic pain and urinary problems. She highlights the complexity of healing from oxalate toxicity, which can be prolonged and involve “healing reactions” such as flares in symptoms, exhaustion, and the necessity of adequate rest.

The interview also touches on the controversial topic of high-dose vitamin C and its relationship to oxalate production, the role of sex hormones in oxalate-related kidney stone risks, and the limitations of probiotics or gut microbiota modifications in fully resolving oxalate toxicity. Sally emphasizes the importance of mineral repletion, particularly through supplementation or mineral baths, to support detoxification and mitigate symptoms.

Finally, Sally talks about her upcoming book Toxic Superfoods, online support groups, and consultations, encouraging people to approach oxalate issues with informed caution, patience, and gradual dietary adjustments.

Highlights

  • 🧪 Oxalates are water-soluble acids and salts that can form harmful crystals in the body, affecting bones, kidneys, joints, and connective tissues.
  • 🥬 Common high-oxalate foods include spinach, chard, beet greens, nuts (especially almonds, cashews, peanuts), quinoa, buckwheat, sweet potatoes, and chocolate.
  • ⚠️ Oxalate toxicity symptoms are diverse and can mimic other chronic conditions: arthritis, migraines, skin rashes, fungal infections, fatigue, and urinary tract irritation.
  • 💊 High-dose vitamin C (oral or IV) can increase oxalate production and worsen crystal formation, cautioning against indiscriminate mega-dosing.
  • 💤 Detoxification from oxalates is a slow process that can cause symptom flares and exhaustion; sleep and rest are critical components of healing.
  • 🧂 Mineral supplementation and mineral baths can support detoxification and alleviate symptoms by replenishing depleted calcium, magnesium, potassium, and citrate.
  • 🚫 Probiotics alone cannot fix oxalate toxicity because the gut ecosystem complexity and environmental factors prevent full restoration of oxalate-degrading bacteria.

Key Insights

  • 🔬 Oxalate Chemistry and Biological Impact: Oxalates exist as oxalic acid or oxalate salts, which can bind to minerals like calcium and magnesium to form tiny insoluble crystals. These crystals precipitate in body tissues, causing inflammation and oxidative stress that undermine cellular function, especially in mitochondria, connective tissue, and glands. This explains the widespread systemic effects beyond just kidney stones, including fatigue, joint pain, and neurological symptoms.

  • 🥗 Dietary Sources and Evolutionary Mismatch: Many popular “healthy” plant foods contain high levels of bioavailable oxalates, which humans are not evolutionarily adapted to consume in large quantities. The presence of oxalates in seeds and fruits serves as plant defense “micro-weaponry” to deter herbivores. Modern diets rich in nuts, dark leafy greens, and gluten-free grains can inadvertently overload the body with oxalates, leading to chronic poisoning symptoms.

  • Oxalate Toxicity Mimics Chronic Illnesses: Symptoms of oxalate poisoning are often mistaken for autoimmune diseases, fibromyalgia, or other chronic syndromes. The immune system reacts to nano-crystals by causing inflammation in joints, connective tissues, bladder, and skin. This inflammation and oxidative stress interfere with cellular signaling, especially calcium signaling, which is critical for cell metabolism and repair.

  • 🛑 Vitamin C Overuse Can Worsen Oxalate Load: Vitamin C metabolizes into oxalic acid, so excessive vitamin C intake—particularly intravenous high-dose therapy—can exacerbate oxalate crystal formation in tissues and veins, causing fibrosis and vascular damage. This is a caution against indiscriminate use of vitamin C supplements without considering oxalate toxicity risks.

  • 🕰️ Healing is a Long-Term Process with Flare-Ups: Oxalate crystals lodged in bones, joints, and organs can take years to clear. Detoxification often triggers immune responses and symptom “healing reactions,” such as rashes, arthritis flares, headaches, and exhaustion. Understanding this pattern helps patients stay patient and avoid discouragement during recovery.

  • 💧 Mineral Balance is Crucial for Prevention and Healing: Oxalates rob the body of essential minerals (calcium, magnesium, potassium), contributing to bone loss, kidney stones, and systemic dysfunction. Supplementing with mineral salts like potassium citrate, magnesium citrate, and using mineral baths can restore mineral balance, alkalinity, and prevent stone formation. Adjusting urinary pH and citrate levels is key to protecting kidney health during oxalate detox.

  • 🌿 Gut Microbiome Interventions Are Insufficient Alone: While gut bacteria can degrade some oxalates, the complexity of the human microbiome and environmental insults prevent the microbiome from fully protecting against oxalate poisoning. Attempts to “fix” oxalate problems solely with probiotics or microbiome therapies have not succeeded clinically, emphasizing that dietary management and mineral support remain foundational.

Additional Context and Practical Takeaways

  • Avoiding high oxalate foods like spinach, nuts, sweet potatoes, and chocolate is the first step for those with symptoms or history of oxalate toxicity.
  • A gradual reduction in oxalate intake is advisable to prevent overwhelming the kidneys with oxalate mobilized from tissues.
  • Incorporating small amounts of certain carbohydrates like low-oxalate vegetables, rice, or sweet potatoes can help modulate the oxalate detoxification process and alleviate exhaustion.
  • Monitoring symptoms such as joint pain, urinary discomfort, skin issues, and neurological disturbances can indicate oxalate load and detox activity.
  • Support groups, educational resources, and professional guidance—such as Sally Norton’s online classes and consultations—are valuable for navigating the complexity of oxalate issues.
  • The upcoming book Toxic Superfoods promises a comprehensive, accessible resource for understanding oxalates and managing related health issues.

The interview is a vital resource for anyone struggling with unexplained chronic symptoms, kidney stones, or those interested in the lesser-known impacts of diet on long-term health. It empowers listeners to take control of their health by recognizing oxalate toxicity as a real and addressable problem with proper knowledge, patience, and support.

Just finished the episode, it was interesting, especially when she went through the different manifestations people experience. I did take a look at pubmed, not too much research outside of stones being published.

I am happy that my zero-carb approach avoids this entirely.

[–] jet@hackertalks.com 3 points 2 weeks ago* (last edited 2 weeks ago) (1 children)

I'd add carbohydrates to the list of culprits. Sugar (raw, refined, etc) and Carbohydrates both end up in the blood stream as glucose, so as far as insulin response goes they are equivalent.

Can someone consume carbohydrates and maintain health? Sure! Is it helping them do so? not so much.

[–] jet@hackertalks.com 1 points 2 weeks ago

One problem with detecting oxalic acid is its not usually in the blood stream (i.e. waiting in the adipocytes)

I've only heard of people doing carnivore reporting Oxalate dumping. Though the protocol seems to be to introduce a small amount of oxalates in the diet to prevent dumping when inconvenient.

[–] jet@hackertalks.com 3 points 2 weeks ago (1 children)

Notes:

most common endocrine disorder in women in the reproductive age, with an estimated prevalence ranging from 6 to 15%

Common signs of PCOS not included in diagnostic cri-teria are represented by insulin resistance, reversal of the FSH/LH ratio and obesity, which is an important clinical feature of PCOS.

it is important to remark that these metabolic abnormalities may also be present in non-obese patients

96% of westerners have impaired metabolic health, its not just the visibly obese!

The ovaries of PCOS patients usually maintain a normal response to insulin.

It's tragic, people with impaired metabolism have elevated insulin levels, but their ovaries are still very insulin sensitive - so the signal is just too strong!

PCOS women present a peculiar dietary pattern, characterised by reduced use of extra-virgin olive oil, legumes, seafood and nuts, a lower amount of complex carbohydrate, fiber, monounsaturated fatty acids, and higher simple carbo- hydrates, total fat and saturated fatty acid, compared to normal women.

it is controversial whether diet composition per se has an effect on reproductive and metabolic outcomes. Blood glucose levels are affected by carbohydrate intake and regulate insulin secretion from the pancreas, so very-low carbohydrates diets may be superior to standard hypocaloric diets in terms of improving fertility, endocrine/metabolic parameters, weight loss and satiety in women with PCOS

That is a UNDERSTATEMENT!

This was a 12 weeks, single-arm study. The outcome measures were body weight, BMI, FBM, LBM, FBM percentage, LBM percentage, glucose, insulin, HOMA-IR, total cholesterol, HDL, LDL, triglycerides, total testosterone, free testosterone, progesterone, estradiol, LH, FSH, DHEAS, LH/FSH ratio, SHBG and Ferriman Gallwey Score.

Anthropometric and body composition measurements revealed an

  • average weight loss of 9.43 kg (pre 81.19 ± 8.44 kg vs post 71.76± 6.66 kg; p < 0.0001)
  • significant reductions (− 3.35) of BMI (pre 28.84 ± 2.10 vspost 25.49 ± 1.69; p < 0.0001)
  • FBM (− 8.29 kg) (pre 27.96 ± 5.11 kg vs post 19.67 ± 3.72 kg; p < 0.0001).
  • LBM absolute value showed a slightly significant decrease (pre53.23 ± 5.02 kg vs post 52.09± 4.60 kg), but its percent- age value was slightly increased (pre 65.74 ± 3.75% vspost 72.71 ± 3.55%; p < 0.0001)
  • VAT showed a very signifi-cant (pre 1750 ± 181.58 grams vs. post 1110,36 ± 189.23;p < 0.0001) decrease
  • waist circumference decreased in a significant manner (pre 100.7 ± 4.81 vs post 96.69 ± 3.82; p = 0.0015)

Not bad for a 3 month study!

Not bad at all!

KDs could be considered, as a nutraceutical therapy aimed to increase insulin sensitivity. The data available in the literature [26, 30–32], although few, confirm the assumption that a KD, correcting hyperinsulinemia and improving body composition, can contribute to the normalization of the clinical picture in PCOS. During fasting or a carbohydrate restriction such as a KD, blood insulin concentration decreases, while glucagon increases to maintain the normal blood glucose level, first through glycogen stores, then through the β-oxidation of fatty acids stored in fat depots. Approximately 3–5 days after a very low carbohydrate diet, when the concentration of KBs begins to grow, hunger considerably decreases, but maintaining a state of well-being [51]

we can be assumed that 12 weeks were not sufficient to observe a decrease in hirsutism scores: the hair cycle, in fact, depending on the body area can last for some months and it is known that pharmacological therapy based on antiandrogens takes from 6 to 12 months to obtain a good reduction of the score.

This is a very important point, hormonal interventions (which a KD is), will take months if not years to fully correct. If it took 30 years to get into a biological state, it might take longer then a 12 week study to correct it.

[–] jet@hackertalks.com 1 points 2 weeks ago* (last edited 2 weeks ago)

There is a established link between elevated insulin levels and PCOS

i.e. Ketogenic - Chapter 3 - Endocrine

3.7.3.3 Polycystic ovarian syndrome and infertility / The insulin connection

“is that the three defining features of PCOS (hyperandrogenism causing masculine features, polycystic ovaries and anovulatory cycles) all reflect the same pathophysiology: too much testosterone, ultimately caused by too much insulin. In other words, too much insulin causes PCOS. Like obesity, PCOS is best understood as a disease of hyperinsulinemia. Although obesity and PCOS do not always occur together, they are both manifestations of an underlying hyperinsulinemia.

The eponymous criterion of PCOS is the presence of multiple cysts in the ovaries, which are derived from the multitude of small follicles. Many women have a few cysts on their ovary, but the sheer number of cysts distinguishes this syndrome from virtually all others. Almost no other much insulin and too much testosterone human disease causes polycystic ovaries. Ultimately, these polycystic ovaries are caused by too.

Both the cysts on the ovaries and the hyperandrogenism are caused by the same underlying problem: too much insulin.

The full book is available on the normal free literature places, but the TLDR is that a diet that reduces insulin levels can be used to treat and reverse PCOS

This is also another more direct (and open) article on the issue: Effects of a ketogenic diet in overweight women with polycystic ovary syndrome - My notes from reading the paper can be found here - https://hackertalks.com/post/13750353

[–] jet@hackertalks.com 2 points 2 weeks ago (3 children)

It's actually really nice to see Bart Kay speaking like a normal human instead of a outrage machine.

 

The mobile khanates are documented eating horse meat and dairy products along their vast nomadic tracts

Renowned for their ability to ride for days and immediately go into battle, this maps to what we understand about modern carnivore - eating only when necessary, skipping meals, very high levels of energy speak to a fat based metabolism

Obviously the Mongols were not exclusive carnivores - they were opportunistic

https://www.historyonthenet.com/what-did-the-mongols-eat

Farming was not possible for the most part, so the most prominent foods in the Mongol diet were meat and milk products such as cheese and yogurt. The Mongols were a nomadic, pastoral culture and they prized their animals: horses, sheep, camels, cattle and goats. As their herds ate up the grass, the Mongols would pack up their gers, tent-like dwellings they lived in, and move their herds to fresher pastures.

Thus, their food groups were predominantly milk products and a variety of meats. While the Mongols appreciated milk products, they didn’t drink fresh milk; instead they fermented milk from mares, making an alcoholic drink known as airag or kumiss. After women finished milking the cattle, goats and sheep, they would process the milk into milk curds, yogurts and airag. The usual beverages were salted tea and airag, fermented mare’s milk.

There are curious theories that the Mongolian decline coincided with a change in their diet https://afe.easia.columbia.edu/mongols/pastoral/masson_smith.pdf

What other famous carnivore civilizations are there?

-3
Dating (hackertalks.com)
 

Hunger and Frequency of Eating are big points of friction when dating someone who doesn't do keto or carnivore.

They are always hungry, I probably was this way before too, but now it's feels like such a huge time sink.

"Let's go to lunch - at the grill!" Great! I love the grill... Literally walking out of the grill "Let's find a snack"... one ice cream later... "Where are we going to have dinner"

I went on a few dates with a vegetarian and they were craving food constantly, even more then the above - every few minutes they had to eat some fruit or something sweet. It was kinda funny until it started to interrupt plans or outings.

I suppose this is just a reflection with how much my relationship with food has changed. Going into markets with a million snack shops seems so out of touch with reality now.

 

I've been doing daily sauna for a month or two now. Feels great ...

80C for 20m every day...

Now the coating in my glasses is melting and everything is a blur of bright lights.

Need to get the rest of the coating off, or get new glasses.

 

No eye diagram of data speed tests, but a neat home project.

 

For 4 weeks, I ate 2,000 calories per day of saturated fat to see what it would do to my cholesterol levels. Spoiler alert: NOTHING. That’s 222 grams of saturated fat per day, and my total and LDL "bad" cholesterol levels remained the same. In this video, I break down why this happened and explain why this isn't just clickbait—it's a science-backed demonstration that will challenge conventional thinking about cholesterol, saturated fat, and cardiovascular health. We'll dive deep into how dietary fat influences cholesterol levels, the role of LDL and HDL, and why it's important to rethink what we know about fat consumption. Prepare to have your curiosity piqued and learn the real science behind these surprising results that appear to defy mainstream perspectives cholesterol.

Chapters 
0:00 – Consuming 2000 Calories from Saturated Fat: What Happened? 
1:02 – How My Baseline Ketogenic Diet Affected My Cholesterol 
1:52 – Switching to a Saturated Fat-Rich Diet: The Details 
3:08 – Surprising Result: My Cholesterol Didn’t Change 
3:25 – Understanding Why My Cholesterol Remained the Same 
8:36 – Why Did I Test Saturated Fat’s Impact on Cholesterol? 
11:01 – How to Test Your Cholesterol Levels at Home
12:39 – The Importance of Metabolic Context in Cholesterol Levels 

LDL cholesterol levels were graphed as ‘baseline normalized,’ i.e. as a ratio of LDL cholesterol at a given timepoint relative to baseline LDL cholesterol level for three reasons (i) Relative change, not absolute value, is most pertinent to the point(s) demonstrated by the experiment (ii) Given Lean Mass Hyper-Responder physiology, my baseline LDL-C on a ketogenic diet – irrespective of the fatty acid composition of that diet – is high. This is not the case when I’m on a diet including carbohydrates, as we’ve published. In prior content, audience has become fixated on absolute numbers relative to their own and attempted to use that as justification for personal choices. The point of the video is a physiologic demonstration, not a “dietary hall pass,” as stated explicitly in the video. (iii) We’ve published the absolute numbers in several papers in the peer-reviewed literature. A collection of papers can be found: cholesterolcode.com/papers

 

I think the zeitgeist would agree that Pizza isn't healthy, it's a junk food

What is a pizza?

  • Bread
  • Cheese
  • Topping (pepperoni)
  • Tomato Sauce

By weight bread is the largest component of pizza.

So which part of this makes it a junk food?

Carnivores would say the Cheese and Meat is fine, and the bread and sauce are making it a junk food

What would have to change on a pizza to make it "healthy"?

 

Sometimes you want to make a post for a community but don't want to reach the ALL audience (niche or controversial communities). Here is a pseudo workaround to achieve this.

  • Make a post
  • Immediately delete the post
  • Wait 12-24 hours
  • Undelete the post

The post will appear in the community at the original creation time and not the undelete time. This avoids the bulk of people who browse all by new (since it will be buried 12 hours in the past). Subscribers will still see the post in their feed

 

Ali Morgan has actually been involved with farming in many different ways for decades in many countries all around the world. Today, she busts a lot of myths about animal and crop farming and shows us the truths about what vegetarianism and veganism does to our beautiful planet. She is also one of the authors of the book: Why Vegans Have Smaller Brains.

https://youtu.be/qO1l43vVUig

00:00 Video Start
1:00 Intro
2:55 Who is Ali Morgan?
5:00 Farming knowledge in the west vs elsewhere
7:25 Nutrition quality in different countries
11:19 Reduction in farming industry
12:33 Automation / Mechanisation
14:25 Loss of rotation damages the environment
18:50 When we lost planet-friendly farming
19:49 Definition of organic farming, vegan farming myths
22:28 Definition of conventional farming
23:30 Soya’s carbon emissions
24:16 Beef doesn’t hurt the amazon
26:06 Crops vs livestock for the environment
36:34 Conventional vs organic vs regenerative farming
45:00 Definition of regenerative farming
48:14 Grain-free poultry?
50:56 Chickens now vs 100 years ago
53:45 How to choose eggs
54:10 What does free range mean?
56:07 Beef breeds & diet-heart hypothesis
59:58 Supermarkets keeping farmers poor, EU subsidies
1:05:10 Milk price factors, cow welfare, pollution from crop farming
1:04:40 Pollution of the environment from farming
1:11:15 Farmers losing money from milk
1:13:20 Mental health / suicides in farming
1:15:44 Further loss of farming industry
1:18:02 Is farming better in other countries?
1:19:50 How do we actually improve food quality?
1:23:49 Buying food in a way that helps farmers
1:26:24 Hormones in meat
1:30:30 Antibiotics in meat
1:37:17 Less meat means more deaths
1:38:15 Financial losses today for farmers
1:43:20 Politicians that understand farming difficulties
1:44:57 Cowspiracy bullshit - the biogenic carbon cycle
1:59:04 Deaths caused by livestock vs. crop farming
2:06:40 What regenerative farming looks like
2:12:58 Pesticides = biodiversity loss, babies with eye deformities 
2:20:09 Can we grow crops without pesticides? Glyphosate
2:25:47 Vegans kill more animals than carnivores
2:34:03 How you can help farmers help us

summerizerSummary

This extensive interview with Allison Morgan, an experienced agricultural expert, challenges common assumptions about farming, diet, and environmental impact, particularly the widely-held belief that vegan or plant-based diets are inherently better for the environment. Morgan draws on decades of firsthand experience in both developed and developing countries to explain the complex relationship between livestock, soil health, crop production, and ecosystem sustainability. She emphasizes that livestock farming—especially ruminants like cattle and sheep—is critical for maintaining soil fertility, carbon sequestration, and biodiversity, while continuous arable crop farming without livestock damages soil and ecosystems. The discussion highlights how modern farming practices, mechanization, and policies have drastically changed traditional mixed crop-livestock systems, often harming soil, environment, and farmer livelihoods. Furthermore, Morgan elucidates the economic challenges farmers face, the mental health crisis within farming communities, and criticisms of oversimplified views on methane emissions from livestock. The interview advocates for more nuanced understanding, better support for farmers, and systemic societal changes to ensure sustainable food systems that balance environmental health, animal welfare, and human economic well-being.

Highlights 🌱 Vegan-only farming without livestock accelerates soil degradation. 🚜 Modern intensive arable farming relies heavily on fossil fuels and agrochemicals, damaging soil health. 🐄 Grazing livestock play a vital role in restoring soil fertility and promoting biodiversity. 🤯 Farmers face high economic pressures, with many barely breaking even or losing money. 💔 Farming professions have some of the highest suicide rates due to stress and financial instability. 💨 Common claims about methane emissions from cattle are often scientifically inaccurate or misleading. 🛒 Direct farm-to-consumer sales and regenerative practices can support better farmer livelihoods and sustainability. Key Insights

🌍 Integrated Crop-Livestock Systems Are Environmentally Essential: Morgan explains that traditional mixed farming systems where livestock graze and manure is returned to fields are crucial for maintaining soil organic matter and fertility. Crops alone, especially in intensive arable rotations, deplete soil carbon and structure, leading to erosion and loss of productivity. This counters the idea that plant-based diets, relying solely on crop farming, are inherently greener. The grass-livestock nexus ensures ecosystem balance and reduces agrochemical dependence.

🔄 Soil Disturbance and Carbon Loss from Arable Farming: Annual crops require plowing and frequent soil disturbance, exposing soil organic matter to oxidation and releasing large amounts of CO₂—estimated as a third of modern carbon emissions from soil. In contrast, perennial grasslands with livestock prevent these losses by maintaining continuous ground cover, which is critical for long-term carbon sequestration and ecosystem resilience.

🐖 Livestock Feed and Sustainability Nuances: Intensive pig and poultry farming in developed countries relies heavily on grain and imported soy, which carries environmental costs and global supply chain concerns. However, ruminants like cattle and sheep mostly graze on permanent pastures, using less grain and soy. These grazing animals fit naturally into ecosystems and can foster biodiversity, unlike more intensive monogastric livestock systems.

💸 Economic Reality of Farming Unveiled: The interview highlights that many farmers, especially beef and sheep producers, operate at a loss without subsidies. Prices dictated by large retailers and supermarkets severely dent farmer incomes, while subsidies often subsidize processors and retailers more than farmers. This economic precarity contributes to mental health crises in farming communities.

💨 Myth Busting Methane Emissions: Contrary to popular media like Cowspiracy, methane from ruminants is part of a short biogenic carbon cycle. Methane breaks down within about a decade into CO₂, which is then reabsorbed by plants, creating a cyclical balance unlike fossil fuel emissions. The demonization of cattle methane grossly exaggerates livestock’s climate impact, overlooks soil carbon capture by grasslands, and neglects wild ruminants.

🐔 Challenges in Regenerative Monogastric Farming: Regenerative poultry and pig farming face inherent challenges because these animals naturally consume more diverse diets, including insects and roots, rather than solely grains. Systems like “mob grazing” where chickens follow cattle and feed on insects in dung offer more sustainable alternatives but cannot eliminate grain feed completely without compromising growth and productivity.

🌿 Agrochemicals, Pesticides, and Biodiversity Collapse: Intensive crop farming applies multiple herbicides, insecticides, and fungicides numerous times a season, resulting in severe declines of beneficial insects, earthworms, and soil biodiversity. This “cocktail of carnage” reduces soil health and critically threatens ecosystem services, including pollination, which grazing livestock systems largely avoid.

Conclusion

The conversation demystifies many myths around veganism, livestock farming, and environmental sustainability by providing a scientifically grounded, practical perspective from a global farming expert. It underlines the urgent need to support farmers financially and psychologically, rethink farming subsidies, and adopt integrated agroecological systems that utilize the synergy between crops and grazing livestock. Only through such nuanced approaches can we ensure resilient ecosystems, nutrient-dense food, animal welfare, and viable rural communities.

 

When your traveling - How do you prepare a travel phone?

  • What information do you sync over?

  • Do you login to accounts before you travel?

  • What is your method of securing your data for cross border traveling?

 

Dr Chaffee presents his paradigm-altering thesis that autoimmune diseases are the result of the body's attack on plant compounds. He presents evidence that removal, as in a carnivore diet, leads to remission of a wide range of autoimmune diseases.

summerizer

Summary

In this presentation at the Regenerate Conference, the speaker challenges the conventional understanding of autoimmune diseases—the idea that these conditions result from the body mistakenly attacking itself due to molecular mimicry or immune dysregulation. Traditionally, autoimmunity is thought to arise when the immune system is sensitized to self-antigens following an environmental trigger, such as a pathogen or toxin, often influenced by genetic predisposition. However, the speaker asserts that this model does not hold up well against clinical and immunological observations. Instead, they propose an alternative explanation focusing on environmental toxins, particularly plant lectins and glyphosate, as causative agents eliciting immune responses. The immune system is not attacking the body indiscriminately but responding appropriately to foreign toxins bound to bodily tissues, which results in collateral tissue damage.

Using celiac disease as a prime example, they explain how gluten, a plant lectin, binds to intestinal lining cells, causing damage that triggers an immune response not against the self but against the offending lectin. Removal of gluten allows intestinal repair despite persistent antibodies, suggesting this is not true autoimmunity. The talk highlights evidence from scientific literature dating back to the early 1990s that supports lectins as major contributors to inflammatory and “autoimmune” diseases, offering a coherent mechanism that accounts for the complex patterns observed in these conditions.

The speaker also discusses how certain populations, like individuals with Down syndrome, show higher prevalence of autoimmune diseases, indicating a genetic susceptibility but emphasizing that genetics alone does not explain the phenomenon. Immunological development processes should prevent self-reactive immune cells from surviving, and if they do not, the immune attack on self-antigens would be severe and relentless, contrary to the typical flare-remission pattern seen clinically.

A critical practical takeaway is that dietary management—specifically removing plant lectins and other toxins—can dramatically improve or even put autoimmune conditions into remission. The carnivore ketogenic diet focused on ruminant meat is highlighted due to its lower lectin and toxin content, and clinical case studies are cited where patients with conditions such as type 1 diabetes and multiple sclerosis experienced significant recovery and symptom remission with this approach. The talk encourages a shift from immune suppression—currently the mainstay of autoimmune disease management and fraught with side effects—to a strategy that removes environmental triggers allowing the immune system to normalize and tissue healing to occur.

Highlights

  • 🌱 Autoimmunity may not be the body attacking itself but an immune response to environmental toxins like plant lectins.
  • 🧬 Genetic predisposition plays a role but cannot fully explain autoimmune disease patterns.
  • 🥩 Diet, especially eliminating plant lectins and consuming ruminant meat, can dramatically improve autoimmune conditions.
  • 🌾 Celiac disease exemplifies how immune response targets toxins stuck to tissues, not the tissues themselves.
  • 🔬 Traditional immunology models suggest true autoimmune cells would mount an unrelenting attack, unlike observed flare-remission patterns.
  • 🧠 Lectins and toxins can cross biological barriers, including the placenta and blood-brain barrier, potentially causing diverse systemic effects.
  • 📉 Case studies show remarkable reversals in diseases like type 1 diabetes and multiple sclerosis through dietary interventions.

Key Insights

  • 🌿 Environmental toxins as primary drivers: The key insight is that many autoimmune-like conditions are possibly triggered by plant lectins and other environmental toxins bound to bodily tissues. This explains why removing offending foods relieves symptoms and why immune response patterns differ from classic infections. Unlike molecular mimicry models, this explanation accounts for the remission-flare cycles and tissue damage observed clinically without attributing the cause to the immune system malfunctioning or attacking self-antigens inherently.

  • 🧠 Immunological central tolerance challenges the autoimmunity theory: During immune cell development in the thymus, autoreactive T cells are generally eliminated to prevent self-attack. For autoimmunity to be genuine, this central tolerance would have to fail catastrophically, which the speaker argues is highly unlikely. Real autoimmune destruction would present as continuous and severe, not remitting. Therefore, the presence of self-reactive antibodies or T cells does not prove the immune system’s intention to attack self, rather it may be responding to foreign substances adhering to self-structures.

  • 🍽️ Dietary intervention as a powerful therapeutic tool: Removing lectins and other toxic exposures via strict dietary controls—exemplified by a carnivore ketogenic diet emphasizing ruminant meat—demonstrates striking clinical improvements. The fermentation process in ruminants reduces toxin levels in their meat, making it safer for consumption. Patients show reductions in symptoms, inflammation, and even tissue healing, as seen in autoimmune diseases like Crohn’s, Hashimoto’s, and type 1 diabetes. This represents a paradigm shift from symptom management by immunosuppression to root-cause elimination.

  • 🧬 Genetics as susceptibility, not sole cause: People with genetic conditions such as Down syndrome have higher rates of autoimmune diseases, confirming some genetic component. However, shared familial environment and diet, as well as other factors, complicate genetics-only explanations. Thus, genetics may predispose but environmental exposures and dietary factors are likely necessary triggers for disease manifestation.

  • 🧪 Lectins damage physiological barriers promoting systemic effects: Lectins’ ability to degrade intestinal microvilli causing leaky gut, and to breach the placental and blood-brain barriers, implicates them in systemic inflammatory and autoimmune-like diseases. This also explains observations such as Parkinson’s disease reduction following vagus nerve removal (a potential entry route for lectins to the brain), reinforcing environmental toxin involvement in disease etiology beyond classical autoimmunity.

  • 📉 Immune suppression insufficient and problematic: Current immunosuppressive therapies for autoimmune diseases treat symptoms but suppress overall immunity, increasing risk for infections and cancers without addressing underlying causes. Identifying and eliminating environmental triggers could avoid these pitfalls, representing a safer, more effective long-term strategy.

  • 🩺 Clinical case evidence supporting reinterpretation: The speaker cites real-world cases where dietary changes led to normalization of immune function and tissue healing, including insulin production restoration in type 1 diabetes patients and multiple sclerosis lesion reduction. These outcomes challenge the prevailing dogma of irreversible autoimmune tissue destruction and encourage further research into diet and toxin-related mechanisms.

Summary Conclusion

This talk advocates for a fundamental rethink of autoimmune diseases, moving away from the entrenched idea of the immune system attacking self. Instead, it promotes the theory that environmental triggers such as lectins and toxins cause tissue-bound antigens that the immune system responds to appropriately. This new understanding better fits observed clinical patterns and opens avenues for safer, causal treatments focusing on dietary and environmental modifications. While genetic predisposition matters, addressing external triggers is key to management and remission of these conditions, suggesting the potential to revolutionize autoimmune disease therapies.

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